Synaptic Plasticity and the Mechanism of Alzheimer's Disease
| Autor Corporativo: | |
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| Otros Autores: | , , |
| Formato: | eBook |
| Lenguaje: | English |
| Publicado: |
Berlin, Heidelberg :
Springer Berlin Heidelberg,
2008.
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| Colección: | Research and Perspectives in Alzheimer's Disease,
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| Materias: | |
| Acceso en línea: | https://doi.org/10.1007/978-3-540-76330-7 |
Tabla de Contenidos:
- Permanence of the Synapse and Molecular Instability
- Cellular Biology of AMPA Receptor Trafficking and Synaptic Plasticity
- Imaging of Experience-Dependent Structural Plasticity in the Mouse Neocortex in vivo
- Synapse Loss, Synaptic Plasticity and the Postsynaptic Density
- Impact of Beta Amyloid on Excitatory Synaptic Transmission and Plasticity
- Quantitative Neuropathology in Alzheimer’s Mouse Models
- Multiple Levels of Synaptic Regulation by NMDA-type Glutamate Receptor in Normal and Disease States
- Soluble Oligomers of the Amyloid ?-Protein: Impair Synaptic Plasticity and Behavior
- Why Alzheimer’s is a Disease of Memory: Synaptic Targeting by Pathogenic A? Oligomers (ADDLs)
- Synaptic Transmission Dynamically Modulates Interstitial Fluid Amyloid-? Levels
- A?-Induced Toxicity Mediated by Caspase Cleavage of the Amyloid Precursor Protein (APP)
- Long-Term Potentiation and A?: Targeting A? Species, Cellular Mechanisms and Putative Receptors
- Genes, Synapses and Autism Spectrum Disorders.