Protein Expression in Down Syndrome Brain
| Autor Corporativo: | |
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| Otros Autores: | |
| Formato: | eBook |
| Lenguaje: | English |
| Publicado: |
Vienna :
Springer Vienna : Imprint: Springer,
2001.
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| Edición: | 1st ed. 2001. |
| Materias: | |
| Acceso en línea: | https://doi.org/10.1007/978-3-7091-6262-0 |
Tabla de Contenidos:
- Decreased alpha-endosulfine, an endogenous regulator of ATP-sensitive potassium channels, in brains from adult Down syndrome patients
- Developmental instability of the cerebellum and its relevance to Down syndrome
- Expression of the multidrug resistance P glycoprotein (Pgp) and multidrug resistance associated protein (MRP1) in Down syndrome brains
- Deterioration of the transcriptional, splicing and elongation machinery in brain of fetal Down Syndrome
- Fetal life in Down Syndrome starts with normal neuronal density but impaired dendritic spines and synaptosomal structure
- Antioxidant proteins in fetal brain: superoxide dismutase-1 (SOD-1) protein is not overexpressed in fetal Down syndrome
- Glial-neurotrophic mechanisms in Down syndrome
- Aberrant expression of dihy-dropyrimidinase related proteins-2, -3 and -4 in fetal Down Syndrome brain
- Decreased protein levels of complex I 30-kDa subunit in fetal Down Syndrome brains
- Selective upregulation of the ubiquitin-proteasome proteolytic pathway proteins, proteasome zeta chain and isopeptidase T in fetal Down syndrome
- Functional genomics of Down Syndrome: a multidisciplinary approach
- Unaltered expression of Fas (CD95/APO-1), Caspase-3, Bcl-2 and Annexins in brain of fetal Down syndrome: evidence against increased apoptosis
- Alteration of caspases and other apoptosis regulatory proteins in Down syndrome
- Expression of apoptosis related proteins: RAIDD, ZIP kinase, Bim/BOD, p21, Bax, Bcl-2 and NF-kB in brains of patients with Down syndrome
- Increased brain protein levels of carbonyl reductase and alcohol dehydrogenase in Down Syndrome and Alzheimer’s disease
- Carbohydrate handling enzymes in fetal Down Syndrome brain
- Changes in nicotinic acetylcholine receptor subunits expression in brain of patients with Down syndrome and Alzheimer’s disease
- Protein levels of human peroxiredoxin subtypes in brains of patients with Alzheimer’s disease and Down Syndrome
- Effects of a single transdermal nicotine dose on cognitive performance in adults with Down syndrome
- The brain in Down syndrome
- Decreased levels of ARPP-19 and PKA in brains of Down syndrome and Alzheimer’s disease
- Increased protein levels of heterogeneous nuclear ribonucleoprotein A2/B1 in fetal Down syndrome brains
- Decreased protein levels of stathmin in adult brains with Down syndrome and Alzheimer’s disease
- Molecular neuropathology of transgenic mouse models of Down syndrome
- Down syndrome patients start early prenatal life with normal cholinergic, monoaminergic and serotoninergic innervation
- Expression profiles of proteins in fetal brain with Down syndrome
- Expression patterns of chaperone proteins in cerebral cortex of the fetus with Down Syndrome: dysregulation of T-complex protein 1
- ß-Amyliod precursor protein, ETS-2 and collagen alpha 1 (VI) chain precursor, encoded on chromosome 21, are not overexpressed in fetal Down syndrome: further evidence against gene dosage effect
- Reduction of nucleoside diphosphate kinase B, Rab GDP-dissociation inhibitor beta and histidine triad nucleotide-binding protein in fetal Down Syndrome brain
- Alteration of gene expression in Down’s syndrome (DS) brains: its significance in neurodegeneration.