Trinucleotide Repeat Protocols
| Autor Corporativo: | |
|---|---|
| Otros Autores: | |
| Formato: | eBook |
| Lenguaje: | English |
| Publicado: |
Totowa, NJ :
Humana Press : Imprint: Humana,
2004.
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| Edición: | 1st ed. 2004. |
| Colección: | Methods in Molecular Biology,
277 |
| Materias: | |
| Acceso en línea: | https://doi.org/10.1385/1592598048 |
Tabla de Contenidos:
- Mouse Models of Triplet Repeat Diseases
- Analysis of Triplet Pepeat DNAs and RNAs
- Analysis of Triplet Repeat Replication by Two-Dimensional Gel Electrophoresis
- Genetic Assays for Triplet Repeat Instability in Yeast
- Detection and Isolation of Trinucleotide Repeat Expansions Using the RED Method
- Analysis of Unstable Triplet Repeats Using Small-Pool Polymerase Chain Reaction
- Real-Time RT-PCR for CTG Repeat-Containing Genes
- Detection and Analysis of Polyglutamine-Containing Proteins and Their Aggregates
- Antibodies Against Huntingtin
- Using Antibodies to Analyze Polyglutamine Stretches
- Solubilization of Aggregates Formed by Expanded Polyglutamine Tract Expression in Cultured Cells
- Establishment of Animal and Cultured Cell Models for Trinucleotide Repeat Diseases
- Caenorhabditis elegans as a Model System for Triplet Repeat Diseases
- Monitoring Aggregate Formation in Organotypic Slice Cultures From Transgenic Mice
- The CGG Repeat and the FMR1 Gene
- Analysis of CTG Repeats Using DM1 Model Mice
- Lentiviral-Mediated Gene Transfer to Model Triplet Repeat Disorders
- Mouse Tissue Culture Models of Unstable Triplet Repeats
- In Vivo Analysis of Trinucleotide Repeat Diseases
- Neurotransmitter Receptor Analysis in Transgenic Mouse Models
- Chromatin Immunoprecipitation Technique for Study of Transcriptional Dysregulation in Intact Mouse Brain
- Techniques for Thick-Section Golgi Impregnation of Formalin-Fixed Brain Tissue
- Assessment of Impaired Proteasomal Function in a Cellular Model of Polyglutamine Diseases
- Assessment of In Vitro and In Vivo Mitochondrial Function in Friedreich’s Ataxia and Huntington’s Disease
- Triplet Repeats and DNA Repair
- Oxidative Damage in Huntington’s Disease.