Protein Misfolding, Aggregation and Conformational Diseases Part B: Molecular Mechanisms of Conformational Diseases /

Detalles Bibliográficos
Autor Corporativo: SpringerLink (Online service)
Otros Autores: Uversky, Vladimir N. (Editor ), Fink, Anthony. (Editor )
Formato: eBook
Lenguaje:English
Publicado: New York, NY : Springer US : Imprint: Springer, 2007.
Edición:1st ed. 2007.
Colección:Protein Reviews ; 6
Materias:
Acceso en línea:https://doi.org/10.1007/978-0-387-36534-3
Tabla de Contenidos:
  • Altered Protein Structure and Enhanced Aggregation/Deposition
  • The Pathogenesis of Alzheimer’s Disease: General Overview
  • Free Radicals, Metal Ions, and A? Aggregation and Neurotoxicity
  • Progress in Understanding the Mechanisms of Neuronal Dysfunction and Degeneration in Parkinson’s Disease
  • ?-Synuclein Aggregation and Parkinson’s Disease
  • Cell Biology of ?-Synuclein: Implications in Parkinson’s Disease and Other Lewy Body Diseases
  • Pathogenesis of Prion Diseases
  • Mammalian Prion Protein
  • The Yeast Prion Proteins Sup35p and Ure2p
  • Immunoglobulin Light Chain and Systemic Light-Chain Amyloidosis
  • Pancreatic Islet Amyloid and Diabetes
  • ?2-Microglobulin and Dialysis-Related Amyloidosis
  • Serum Amyloid A and AA Amyloidosis
  • Point Mutations and Enhanced Protein Deposition
  • Transthyretin and the Transthyretin Amyloidoses
  • Human Lysozyme
  • Serpins and the Diversity of Conformational Diseases
  • Altered Protein Structure and Impaired Function
  • Human Copper-Zinc Superoxide Dismutase and Familial Amyotrophic Lateral Sclerosis
  • Understanding the Effects of Cancer-Associated Mutations in the Tumor Suppressor Protein p53: Structural Consequences of Mutations and Possible Ways of Rescuing Oncogenic Mutants
  • Changes in Supramolecular Structure
  • Protein Aggregation in Muscle Fibers and Respective Neuromuscular Disorders
  • Muscular Dystrophies and Protein Mutations
  • The Functional Consequences of Dystrophin Deficiency in Skeletal Muscles
  • Eye Lens Proteins and Cataracts
  • Altered Protein Structure and Changes in Cellular/Nuclear Function
  • Glutamine/Asparagine-Rich Regions in Proteins and Polyglutamine Diseases
  • Mechanistic Insights into the Polyglutamine Ataxias
  • Molecular Pathogenesis of the Polyglutamine Disease: Spinal and Bulbar Muscular Atrophy
  • Post-Translational Modification and Protein Conformational Diseases
  • Protein Glycation and Cataract: A Conformational Disease
  • Defective Glycosylation and Muscular Dystrophies.